CSF p-tau increase in response to Aβ-type and Danish-type cerebral amyloidosis and in the absence of neurofibrillary tangles.

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第一作者： Stephan A,Kaeser

第一单位： Department of Cellular Neurology, Hertie Institute for Clinical Brain Research, University of Tübingen, 72076, Tübingen, Germany. stephan.kaeser@uni-tuebingen.de.;DZNE, German Center for Neurodegenerative Diseases, 72076, Tübingen, Germany. stephan.kaeser@uni-tuebingen.de.

作者： Stephan A,Kaeser ^[1] ; Lisa M,Häsler ^[2] ; Marius,Lambert ^[2] ; Carina,Bergmann ^[2] ; Astrid,Bottelbergs ^[3] ; Clara,Theunis ^[3] ; Marc,Mercken ^[3] ; Mathias,Jucker ^[4]

作者单位： Department of Cellular Neurology, Hertie Institute for Clinical Brain Research, University of Tübingen, 72076, Tübingen, Germany. stephan.kaeser@uni-tuebingen.de.;DZNE, German Center for Neurodegenerative Diseases, 72076, Tübingen, Germany. stephan.kaeser@uni-tuebingen.de. ^[1] Department of Cellular Neurology, Hertie Institute for Clinical Brain Research, University of Tübingen, 72076, Tübingen, Germany.;DZNE, German Center for Neurodegenerative Diseases, 72076, Tübingen, Germany. ^[2] Neuroscience Department, Janssen Research and Development, Beerse, Belgium. ^[3] Department of Cellular Neurology, Hertie Institute for Clinical Brain Research, University of Tübingen, 72076, Tübingen, Germany. mathias.jucker@uni-tuebingen.de.;DZNE, German Center for Neurodegenerative Diseases, 72076, Tübingen, Germany. mathias.jucker@uni-tuebingen.de. ^[4]