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Neuropathological Mechanisms of β-N-Methylamino-L-Alanine (BMAA) with a Focus on Iron Overload and Ferroptosis.

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第一作者： Hamed,Kazemi Shariat Panahi

第一单位： Neuroinflammation Group, Macquarie Medical School, Faculty of Medicine, Health and Human Sciences, Macquarie University, Sydney, NSW, Australia.

作者： Hamed,Kazemi Shariat Panahi ^[1] ; Mona,Dehhaghi ^[2] ; Benjamin,Heng ^[1] ; Darius J R,Lane ^[3] ; Ashley I,Bush ^[3] ; Gilles J,Guillemin ^[4] ; Vanessa X,Tan ^[5]

作者单位： Neuroinflammation Group, Macquarie Medical School, Faculty of Medicine, Health and Human Sciences, Macquarie University, Sydney, NSW, Australia. ^[1] Neuroinflammation Group, Macquarie Medical School, Faculty of Medicine, Health and Human Sciences, Macquarie University, Sydney, NSW, Australia.;PANDIS.Org, Bendigo, Australia.;Department of Microbial Biotechnology, School of Biology and Centre of Excellence in Phylogeny of Living Organisms, College of Science, University of Tehran, Tehran, Iran. ^[2] Melbourne Dementia Research Centre, The Florey Institute of Neuroscience and Mental Health, The University of Melbourne, Parkville, VIC, Australia. ^[3] Neuroinflammation Group, Macquarie Medical School, Faculty of Medicine, Health and Human Sciences, Macquarie University, Sydney, NSW, Australia. gilles.guillemin@mq.edu.au.;PANDIS.Org, Bendigo, Australia. gilles.guillemin@mq.edu.au. ^[4] Neuroinflammation Group, Macquarie Medical School, Faculty of Medicine, Health and Human Sciences, Macquarie University, Sydney, NSW, Australia.;PANDIS.Org, Bendigo, Australia. ^[5]

关键词 Excitotoxicity Ferroptosis Iron overload TAR DNA-binding protein 43 (TDP-43)Tauopathy β-N-methylamino-L-alanine (BMAA)

医学主题词氨基酸类, 二氨基(Amino Acids, Diamino);氰基细菌(Cyanobacteria);人类(Humans);铁超负荷(Iron Overload);神经变性疾病(Neurodegenerative Diseases);神经毒素类(Neurotoxins)

DOI 10.1007/s12640-021-00455-6

PMID 35023054

发布时间 2024-07-15

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