The novel ER stress inducer Sec C triggers apoptosis by sulfating ER cysteine residues and degrading YAP <i>via</i> ER stress in pancreatic cancer cells.

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作者： Junxia,Wang ^[1] ; Minghua,Chen ^[1] ; Mengyan,Wang ^[1] ; Wenxia,Zhao ^[1] ; Conghui,Zhang ^[1] ; Xiujun,Liu ^[1] ; Meilian,Cai ^[1] ; Yuhan,Qiu ^[1] ; Tianshu,Zhang ^[1] ; Huimin,Zhou ^[1] ; Wuli,Zhao ^[1] ; Shuyi,Si ^[1] ; Rongguang,Shao ^[1]

作者单位： Key Laboratory of Antibiotic Bioengineering, Ministry of Health, Laboratory of Oncology, Institute of Medicinal Biotechnology, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, China. ^[1]

关键词 ER stress inducer Fast shrinkage Lipid droplet formation Pancreatic cancer Resistance Secoemestrin C YAP degradation YAP destruction complex

DOI 10.1016/j.apsb.2021.07.004

PMID 35127381

发布时间 2024-04-05

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Acta pharmaceutica Sinica. B

2022年12卷1期

210-227页

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The novel ER stress inducer Sec C triggers apoptosis by sulfating ER cysteine residues and degrading YAP <i>via</i> ER stress in pancreatic cancer cells.

Acta pharmaceutica Sinica. B

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Acta pharmaceutica Sinica. B

相关期刊

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The novel ER stress inducer Sec C triggers apoptosis by sulfating ER cysteine residues and degrading YAP <i>via</i> ER stress in pancreatic cancer cells.

Acta pharmaceutica Sinica. B

相似文献

Acta pharmaceutica Sinica. B

相关期刊

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