Interactomic inhibition of Eomes in the nucleus alleviates EAE via blocking the conversion of Th17 cells into non-classic Th1 cells.

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第一作者： Bo-Young,Shin

第一单位： Department of Biotechnology, Yonsei University College of Life Science and Biotechnology, Seoul, Republic of Korea.

作者： Bo-Young,Shin ^[1] ; Su-Hyeon,Lee ^[1] ; Yuna,Kim ^[1] ; Jaekyeung,An ^[1] ; Tae-Yoon,Park ^[2] ; Sang-Kyou,Lee ^[3]

作者单位： Department of Biotechnology, Yonsei University College of Life Science and Biotechnology, Seoul, Republic of Korea. ^[1] Molecular Neurobiology Laboratory, Department of Psychiatry and McLean Hospital, Harvard Medical School, Belmont, MA, USA. ^[2] Department of Biotechnology, Yonsei University College of Life Science and Biotechnology, Seoul, Republic of Korea.;Good T cells, Inc, Seoul, Republic of Korea. ^[3]

关键词 T-cell subset differentiation Th17 experimental autoimmune encephalomyelitis intranuclear delivery of the protein non-classic Th1

主题词动物(Animals);细胞分化(Cell Differentiation);脑脊髓炎, 自身免疫性, 实验性(Encephalomyelitis, Autoimmune, Experimental);炎症(Inflammation);Th1细胞(Th1 Cells);Th17细胞(Th17 Cells)

DOI 10.1080/25785826.2022.2031812

PMID 35130134

发布时间 2022-06-24

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Immunological medicine

2022年45卷2期

119-127页

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Interactomic inhibition of Eomes in the nucleus alleviates EAE via blocking the conversion of Th17 cells into non-classic Th1 cells.

Immunological medicine

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Immunological medicine

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Interactomic inhibition of Eomes in the nucleus alleviates EAE via blocking the conversion of Th17 cells into non-classic Th1 cells.

Immunological medicine

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Immunological medicine

相关期刊

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