Auto-inflammation and auto-immunity pathways are associated with emergence of BOS in pediatric lung transplantation.

导出 OA

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作者： Carol K,Conrad ^[1] ; Haley,Hedlin ^[2] ; Hyunsook,Chin ^[3] ; Don,Hayes ^[4] ; Peter S,Heeger ^[5] ; Albert,Faro ^[6] ; Samuel,Goldfarb ^[7] ; Ernestina,Melicoff-Portillo ^[8] ; Mohanakumar,Thalachallour ^[9] ; Jonah,Odim ^[10] ; Marc,Schecter ^[11] ; Gregory A,Storch ^[12] ; Gary A,Visner ^[13] ; Nikki M,Williams ^[10] ; Karen,Kesler ^[3] ; Lara,Danziger-Isakov ^[3] ; Stuart C,Sweet ^[11]

作者单位： Department of PediatricsStanford University School of Medicine, Palo Alto, California, USA. ^[1] Quantitative Sciences Unit, Stanford University School of Medicine, Stanford, California, USA. ^[2] Rho Federal Systems, Durham, North Carolina, USA. ^[3] Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, USA. ^[4] Department of Medicine, Translational Transplant Research Center, Icahn School of Medicine at Mount Sinai, New York, New York, USA. ^[5] Cystic Fibrosis Foundation, Bethesda, Maryland, USA. ^[6] Masonic Children's Hospital, University of Minnesota, Minneapolis, Minnesota, USA. ^[7] Texas Children's Hospital, Houston, Texas, USA. ^[8] Norton Thoracic Institute, St. Joseph's Hospital and Medical Center, Phoenix, Arizona, USA. ^[9] National Institutes of Health, NIAID, Bethesda, Maryland, USA. ^[10] University of Florida, Gainsville, Florida, USA. ^[11] Washington University School of Medicine, St. Louis, Missouri, USA. ^[12] Boston Children's Hospital, Boston, Massachusetts, USA. ^[13]

关键词 BOS CLAD auto-inflammation pediatric lung transplant