Tourniquet-induced lower limb ischemia/reperfusion reduces mitochondrial function by decreasing mitochondrial biogenesis in acute kidney injury in mice.

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作者： Balamurugan,Packialakshmi ^[1] ; Ian J,Stewart ^[2] ; David M,Burmeister ^[1] ; Yuanyi,Feng ^[1] ; Dennis P,McDaniel ^[3] ; Kevin K,Chung ^[4] ; Xiaoming,Zhou ^[1]

作者单位： Department of Medicine, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA. ^[1] The Henry Jackson M. Foundation for the Advancement of Military Medicine, Bethesda, Maryland, USA. ^[2] Department of Biochemistry, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA. ^[3] Biomedical Instrumentation Center, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA. ^[4]

关键词 autophagy ischemia mitochondrial complex mitochondrial oxidative stress mitophagy

主题词急性肾损伤(Acute Kidney Injury);动物(Animals);HSP27热休克蛋白质类(HSP27 Heat-Shock Proteins);后肢(Hindlimb);缺血(Ischemia);缺血预处理(Ischemic Preconditioning);男(雄)性(Male);小鼠(Mice);线粒体, 肌(Mitochondria, Muscle);氧化性应激(Oxidative Stress);蛋白激酶类(Protein Kinases);泛素蛋白连接酶类(Ubiquitin-Protein Ligases)

DOI 10.14814/phy2.15181

PMID 35146957

发布时间 2022-10-05

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Physiological reports

2022年10卷3期

e15181页

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Tourniquet-induced lower limb ischemia/reperfusion reduces mitochondrial function by decreasing mitochondrial biogenesis in acute kidney injury in mice.

Physiological reports

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Physiological reports

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Tourniquet-induced lower limb ischemia/reperfusion reduces mitochondrial function by decreasing mitochondrial biogenesis in acute kidney injury in mice.

Physiological reports

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Physiological reports

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