Inhibition of TRPA1 Attenuates Oxidative Stress-induced Damage After Traumatic Brain Injury via the ERK/AKT Signaling Pathway.

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第一作者： Xin-Jie,Yang

第一单位： The Translational Research Institute for Neurological Disorders, Department of Neurosurgery, The First Affiliated Hospital (Yijishan Hospital), Wannan Medical College, Wuhu, China.

作者： Xin-Jie,Yang ^[1] ; Shizhang,Ling ^[1] ; Meng-Liang,Zhou ^[2] ; Hong-Ji,Deng ^[3] ; Min,Qi ^[1] ; Xi-Lin,Liu ^[1] ; Cheng,Zhen ^[4] ; Yun-Xiao,Chen ^[1] ; Xi-Ran,Fan ^[1] ; Ze-Yu,Wu ^[1] ; Feng-Chun,Ma ^[1] ; Jun,Rong ^[1] ; Guang-Fu,Di ^[5] ; Xiao-Chun,Jiang ^[6]

作者单位： The Translational Research Institute for Neurological Disorders, Department of Neurosurgery, The First Affiliated Hospital (Yijishan Hospital), Wannan Medical College, Wuhu, China. ^[1] Department of Neurosurgery, Jinling Hospital, School of Medicine, Nanjing University, Jiangsu, China. ^[2] Department of Neurosurgery, The First Affiliated Hospital, Guangxi Medical University, Nanning, China. ^[3] Cell Electrophysiology Laboratory, Wannan Medical College, Wuhu, China. ^[4] The Translational Research Institute for Neurological Disorders, Department of Neurosurgery, The First Affiliated Hospital (Yijishan Hospital), Wannan Medical College, Wuhu, China. Electronic address: wuhusjwk@163.com. ^[5] The Translational Research Institute for Neurological Disorders, Department of Neurosurgery, The First Affiliated Hospital (Yijishan Hospital), Wannan Medical College, Wuhu, China. Electronic address: jiangxiaochun2019@hotmail.com. ^[6]

关键词 apoptosis reactive oxidative species transient receptor potential ankyrin 1 traumatic brain injury