CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses.

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作者： Yu,Chen ^[1] ; Qiaomei,Cai ^[2] ; Chaohu,Pan ^[3] ; Wancheng,Liu ^[2] ; Lili,Li ^[3] ; Junxiao,Liu ^[1] ; Meiling,Gao ^[2] ; Xiaorong,Li ^[3] ; Liguo,Wang ^[2] ; Yu,Rao ^[3] ; Heng,Yang ^[2] ; Genhong,Cheng ^[3]

作者单位： Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and School of Basic Medicine, Peking Union Medical College, Beijing, P.R. China. ^[1] Institute of Systems Medicine, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, P.R. China. ^[2] Suzhou Institute of Systems Medicine, Suzhou, Jiangsu, P.R. China. ^[3] MOE Key Laboratory of Protein Sciences, School of Pharmaceutical Sciences, MOE Key Laboratory of Bioorganic Phosphorus Chemistry and Chemical Biology, Tsinghua University, Beijing, P.R. China. ^[4] Department of Microbiology, Immunology & Molecular Genetics, University of California Los Angeles, Los Angeles, California. ^[5]

主题词动物(Animals);细胞周期蛋白依赖激酶2(Cyclin-Dependent Kinase 2);内源性逆转录病毒(Endogenous Retroviruses);小鼠(Mice);磷酰化(Phosphorylation);视网膜母细胞瘤蛋白质(Retinoblastoma Protein)

DOI 10.1158/2326-6066.CIR-21-0806

PMID 35181784

发布时间 2022-06-01

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Cancer immunology research

2022年10卷4期

525-539页

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CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses.

Cancer immunology research

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CDK2 Inhibition Enhances Antitumor Immunity by Increasing IFN Response to Endogenous Retroviruses.

Cancer immunology research

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Cancer immunology research

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