Immune cell multiomics analysis reveals contribution of oxidative phosphorylation to B-cell functions and organ damage of lupus.

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作者： Yusuke,Takeshima ^[1] ; Yukiko,Iwasaki ^[2] ; Masahiro,Nakano ^[3] ; Yuta,Narushima ^[4] ; Mineto,Ota ^[1] ; Yasuo,Nagafuchi ^[5] ; Shuji,Sumitomo ^[1] ; Tomohisa,Okamura ^[2] ; Keith,Elkon ^[1] ; Kazuyoshi,Ishigaki ^[2] ; Akari,Suzuki ^[1] ; Yuta,Kochi ^[1] ; Kazuhiko,Yamamoto ^[2] ; Keishi,Fujio ^[6]

作者单位： Department of Allergy and Rheumatology, The University of Tokyo, Tokyo, Japan. ^[1] Department of Functional Genomics and Immunological Diseases, The University of Tokyo, Tokyo, Japan. ^[2] Department of Allergy and Rheumatology, The University of Tokyo, Tokyo, Japan fujiok-int@h.u-tokyo.ac.jp yu_nyan@saitama-med.ac.jp. ^[3] Department of Palliative Medicine, Saitama Medical University, Saitama, Japan. ^[4] Research Division, Chugai Pharmaceutical Co Ltd, Kamakura, Japan. ^[5] Division of Rheumatology, University of Washington, Seattle, Washington, USA. ^[6] Laboratory for Autoimmune Diseases, Center for Integrative Medical Sciences, Riken Yokohama Institute, Yokohama, Japan. ^[7] Department of Genomic Function and Diversity, Tokyo Medical and Dental University, Tokyo, Japan. ^[8]

关键词 B-lymphocytes autoimmunity lupus erythematosus, systemic

主题词 B-淋巴细胞(B-Lymphocytes);人类(Humans);干扰素Ⅰ型(Interferon Type I);红斑狼疮, 系统性(Lupus Erythematosus, Systemic);氧化磷酸化(Oxidative Phosphorylation)

DOI 10.1136/annrheumdis-2021-221464

PMID 35236659

发布时间 2022-07-22

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