Sorafenib-Induced Autophagy Promotes Glycolysis by Upregulating the p62/HDAC6/HSP90 Axis in Hepatocellular Carcinoma Cells.

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第一作者： Xiaoyu,Yan

第一单位： China-Japan Union Hospital, Jilin University, Changchun, China.

作者： Xiaoyu,Yan ^[1] ; Rui,Tian ^[2] ; Jicheng,Sun ^[1] ; Yuanxin,Zhao ^[2] ; Buhan,Liu ^[1] ; Jing,Su ^[2] ; Minghua,Li ^[2] ; Wei,Sun ^[2] ; Xuesong,Xu ^[3]

作者单位： China-Japan Union Hospital, Jilin University, Changchun, China. ^[1] Key Laboratory of Pathobiology, Ministry of Education, Department of Pathophysiology, College of Basic Medical Sciences, Jilin University, Changchun, China. ^[2] Department of Molecular Biology, College of Basic Medical Sciences Jilin University, Changchun, China. ^[3] Jilin Province Zebrafish Genetic Engineering Laboratory, Jilin Province Development and Reform Commission, Jilin, China. ^[4]

关键词 HDAC6 autophagy glycolysis hepatocellular carcinoma p62 sorafenib

DOI 10.3389/fphar.2021.788667

PMID 35250553

发布时间 2022-03-08

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Frontiers in pharmacology

2021年12卷

788667页

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Sorafenib-Induced Autophagy Promotes Glycolysis by Upregulating the p62/HDAC6/HSP90 Axis in Hepatocellular Carcinoma Cells.

Frontiers in pharmacology

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Frontiers in pharmacology

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Sorafenib-Induced Autophagy Promotes Glycolysis by Upregulating the p62/HDAC6/HSP90 Axis in Hepatocellular Carcinoma Cells.

Frontiers in pharmacology

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Frontiers in pharmacology

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