High glucose-mediated VPS26a down-regulation dysregulates neuronal amyloid precursor protein processing and tau phosphorylation.
第一作者:
Chang Woo,Chae
第一单位:
Department of Veterinary Physiology, College of Veterinary Medicine, Research Institute for Veterinary Science, and BK21 Four Future Veterinary Medicine Leading Education & Research Center, Seoul National University, Seoul, Republic of Korea.
作者:
主题词
阿尔茨海默病(Alzheimer Disease);淀粉样β肽类(Amyloid beta-Peptides);淀粉样β蛋白前体(Amyloid beta-Protein Precursor);动物(Animals);糖尿病, 实验性(Diabetes Mellitus, Experimental);减量调节(Down-Regulation);葡萄糖(Glucose);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);磷酰化(Phosphorylation);膜泡运输蛋白质类(Vesicular Transport Proteins)
DOI
10.1111/bph.15836
PMID
35297035
发布时间
2022-07-18
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British journal of pharmacology
3934-3950页
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