Engineering new metabolic pathways in isolated cells for the degradation of guanidinoacetic acid and simultaneous production of creatine.

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作者： Marzia,Bianchi ^[1] ; Luigia,Rossi ^[1] ; Francesca,Pierigè ^[2] ; Pietro,De Angeli ^[1] ; Mattia Paolo,Aliano ^[3] ; Claudia,Carducci ^[1] ; Emanuele,Di Carlo ^[4] ; Tiziana,Pascucci ^[4] ; Francesca,Nardecchia ^[5] ; Vincenzo,Leuzzi ^[6] ; Mauro,Magnani ^[7]

作者单位： Department of Biomolecular Sciences, University of Urbino Carlo Bo, 61029, Urbino, Italy. ^[1] EryDel, Via Antonio Meucci 3, 20091 Bresso, Milan, Italy. ^[2] Center for Ophthalmology, Institute for Ophthalmic Research, University of Tübingen, Tübingen, Germany. ^[3] Department of Experimental Medicine, Sapienza University, 00161 Rome, Italy. ^[4] Department of Psychology and "Daniel Bovet" Center, Sapienza University, 00184 Rome, Italy. ^[5] Istituto di Ricovero e Cura a Carattere Scientifico Fondazione Santa Lucia, 00142 Rome, Italy. ^[6] Division of Child Neurology and Psychiatry, Department of Human Neuroscience, Sapienza University, 00185 Rome, Italy. ^[7]

关键词 GAMT deficiency RBC loading creatine deficit guanidinoacetate methyltransferase (GAMT)metabolic engineering methionine adenosyl transferase

DOI 10.1016/j.omtm.2022.02.007

PMID 35317049

发布时间 2022-03-24

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Molecular therapy. Methods & clinical development

2022年25卷

26-40页

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Engineering new metabolic pathways in isolated cells for the degradation of guanidinoacetic acid and simultaneous production of creatine.

Molecular therapy. Methods & clinical development

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Molecular therapy. Methods & clinical development

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Engineering new metabolic pathways in isolated cells for the degradation of guanidinoacetic acid and simultaneous production of creatine.

Molecular therapy. Methods & clinical development

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Molecular therapy. Methods & clinical development

相关期刊

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