Homologous recombination deficiency is inversely correlated with microsatellite instability and identifies immunologically cold tumors in most cancer types.

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作者： Jan,Budczies ^[1] ; Klaus,Kluck ^[2] ; Susanne,Beck ^[3] ; Iordanis,Ourailidis ^[4] ; Michael,Allgäuer ^[1] ; Michael,Menzel ^[1] ; Daniel,Kazdal ^[1] ; Lukas,Perkhofer ^[1] ; Alexander,Kleger ^[1] ; Peter,Schirmacher ^[1] ; Thomas,Seufferlein ^[4] ; Albrecht,Stenzinger ^[3]

作者单位： Institute of Pathology, University Hospital Heidelberg, Heidelberg, Germany. ^[1] German Cancer Consortium (DKTK), Heidelberg, Germany. ^[2] Centers for Personalized Medicine (ZPM), Heidelberg and Ulm Partner Sites, Germany. ^[3] German Center for Lung Research (DZL), Heidelberg, Germany. ^[4] Department of Internal Medicine 1, University Hospital Ulm, Ulm, Germany. ^[5]

关键词 HRD MSI PARP inhibitors T-cell inflamed gene expression profile homologous recombination deficiency immune cell populations microsatellite instability tumor mutational burden

主题词女(雌)性(Female);人类(Humans);微卫星不稳定性(Microsatellite Instability);卵巢肿瘤(Ovarian Neoplasms);肿瘤微环境(Tumor Microenvironment)

DOI 10.1002/cjp2.271

PMID 35384413

发布时间 2022-08-03

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The journal of pathology. Clinical research

2022年8卷4期

371-382页

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Homologous recombination deficiency is inversely correlated with microsatellite instability and identifies immunologically cold tumors in most cancer types.

The journal of pathology. Clinical research

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The journal of pathology. Clinical research

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Homologous recombination deficiency is inversely correlated with microsatellite instability and identifies immunologically cold tumors in most cancer types.

The journal of pathology. Clinical research

相似文献

The journal of pathology. Clinical research

相关期刊

检索历史清除