Urinary CC16, a potential indicator of lung integrity and inflammation, increases in children after short-term exposure to PM<sub>2.5</sub>/PM<sub>10</sub> and is driven by the CC16 38GG genotype.

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作者： Sarah J D,Nauwelaerts ^[1] ; Nina,Van Goethem ^[2] ; Berta Tenas,Ureña ^[3] ; Koen,De Cremer ^[4] ; Alfred,Bernard ^[5] ; Nelly D,Saenen ^[6] ; Tim S,Nawrot ^[7] ; Nancy H C,Roosens ^[3] ; Sigrid C J,De Keersmaecker ^[8]

作者单位： Transversal Activities in Applied Genomics, Sciensano, Brussels, Belgium; Centre for Toxicology and Applied Pharmacology, University Catholique de Louvain, Woluwe, Brussels, Belgium. ^[1] Department of Epidemiology and Public Health, Sciensano, Brussels, Belgium; Department of Epidemiology and Biostatistics, Institut de Recherche Expérimentale et Clinique, Faculty of Public Health, Université catholique de Louvain, Belgium. ^[2] Transversal Activities in Applied Genomics, Sciensano, Brussels, Belgium. ^[3] Platform Chromatography and Mass Spectrometry, Sciensano, Brussels, Belgium. ^[4] Centre for Toxicology and Applied Pharmacology, University Catholique de Louvain, Woluwe, Brussels, Belgium. ^[5] Centre for Environmental Sciences, Hasselt University, Diepenbeek, Belgium. ^[6] Centre for Environmental Sciences, Hasselt University, Diepenbeek, Belgium; Department of Public Health and Primary Care, KU Leuven, Leuven, Belgium. ^[7] Transversal Activities in Applied Genomics, Sciensano, Brussels, Belgium. Electronic address: Sigrid.dekeersmaecker@sciensano.be. ^[8]

关键词 CC16 Children Genotype PM Respiratory health Urinary biomarkers