Aberrant DNA repair as a potential contributor for the clonal evolution in subsets of anaplastic thyroid carcinomas arising through dedifferentiation: implications for future therapeutic algorithms?

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作者： Carl Christofer,Juhlin ^[1]

作者单位： Department of Oncology-Pathology, Karolinska Institutet, Stockholm SE-17176, Sweden. ^[1] Department of Pathology and Cytology, Karolinska University Hospital, Stockholm SE-17176, Sweden. ^[2]

关键词 DNA repair P53 anaplastic thyroid carcinoma clone dedifferentiation mismatch repair thyroid carcinoma treatment

DOI 10.20517/cdr.2020.66

PMID 35582231

发布时间 2022-05-19

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Cancer drug resistance (Alhambra, Calif.)

2020年3卷4期

992-1000页

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Aberrant DNA repair as a potential contributor for the clonal evolution in subsets of anaplastic thyroid carcinomas arising through dedifferentiation: implications for future therapeutic algorithms?

Cancer drug resistance (Alhambra, Calif.)

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Cancer drug resistance (Alhambra, Calif.)

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Aberrant DNA repair as a potential contributor for the clonal evolution in subsets of anaplastic thyroid carcinomas arising through dedifferentiation: implications for future therapeutic algorithms?

Cancer drug resistance (Alhambra, Calif.)

相似文献

Cancer drug resistance (Alhambra, Calif.)

相关期刊

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