Functional analysis reveals that Tinagl1 is required for normal muscle development in mice through the activation of ERK signaling.

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作者： Yoriko,Sato ^[1] ; Keisuke,Kawashima ^[2] ; Emiko,Fukui ^[2] ; Hiromichi,Matsumoto ^[2] ; Fumiaki,Yoshizawa ^[2] ; Yusuke,Sato ^[3]

作者单位： Department of Animal Science, School of Agriculture, Tokai University, Kumamoto 8628652, Japan. ^[1] Department of Agrobiology and Bioresources, School of Agriculture, Utsunomiya University, Tochigi, 3218505, Japan. ^[2] Department of Animal Science, School of Agriculture, Tokai University, Kumamoto 8628652, Japan. Electronic address: sy163653@tsc.u-tokai.ac.jp. ^[3]

关键词 Capillary ERK signaling Myopathy Skeletal muscle Tinagl1

主题词动物(Animals);内皮细胞(Endothelial Cells);女(雌)性(Female);脂笼蛋白质类(Lipocalins);MAP激酶信号系统(MAP Kinase Signaling System);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);肌肉发育(Muscle Development);肌, 骨骼(Muscle, Skeletal);成肌细胞(Myoblasts);肿瘤蛋白质类(Neoplasm Proteins);转录因子(Transcription Factors)

DOI 10.1016/j.bbamcr.2022.119294

PMID 35597451

发布时间 2022-06-22

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Biochimica et biophysica acta. Molecular cell research

2022年1869卷9期

119294页

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Functional analysis reveals that Tinagl1 is required for normal muscle development in mice through the activation of ERK signaling.

Biochimica et biophysica acta. Molecular cell research

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Biochimica et biophysica acta. Molecular cell research

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Functional analysis reveals that Tinagl1 is required for normal muscle development in mice through the activation of ERK signaling.

Biochimica et biophysica acta. Molecular cell research

相似文献

Biochimica et biophysica acta. Molecular cell research

相关期刊

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