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Tim-1 mucin domain-mutant mice display exacerbated atherosclerosis.

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第一作者： Hidde,Douna

第一单位： Division of BioTherapeutics, LACDR, Leiden University, Einsteinweg 55, 2333CC, Leiden, the Netherlands.

作者： Hidde,Douna ^[1] ; Virginia,Smit ^[1] ; Gijs H M,van Puijvelde ^[1] ; Mate G,Kiss ^[2] ; Christoph J,Binder ^[2] ; Llze,Bot ^[1] ; Vijay K,Kuchroo ^[3] ; Andrew H,Lichtman ^[4] ; Johan,Kuiper ^[1] ; Amanda C,Foks ^[5]

作者单位： Division of BioTherapeutics, LACDR, Leiden University, Einsteinweg 55, 2333CC, Leiden, the Netherlands. ^[1] Department of Laboratory Medicine, Medical University of Vienna, Vienna, 1090, Austria; CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria. ^[2] Department of Neurology, Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School, Boston, MA, 02115, USA. ^[3] Department of Pathology, Brigham and Women's Hospital, Boston, MA, 02115, USA. ^[4] Division of BioTherapeutics, LACDR, Leiden University, Einsteinweg 55, 2333CC, Leiden, the Netherlands. Electronic address: a.c.foks@lacdr.leidenuniv.nl. ^[5]

关键词 Atherosclerosis Cardiovascular disease Immunity Inflammation Tim-1

医学主题词动物(Animals);动脉粥样硬化(Atherosclerosis);白细胞介素10(Interleukin-10);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);黏蛋白类(Mucins)

DOI 10.1016/j.atherosclerosis.2022.05.017

PMID 35636144

发布时间 2022-07-14

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