Distinct stromal and immune cell interactions shape the pathogenesis of rheumatoid and psoriatic arthritis.

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第一作者： Achilleas,Floudas

第一单位： Molecular Rheumatology, Clinical Medicine, Trinity Biomedical Science Institute, Dublin, Ireland.

作者： Achilleas,Floudas ^[1] ; Conor M,Smith ^[2] ; Orla,Tynan ^[3] ; Nuno,Neto ^[1] ; Vinod,Krishna ^[2] ; Sarah M,Wade ^[4] ; Megan,Hanlon ^[5] ; Clare,Cunningham ^[1] ; Viviana,Marzaioli ^[2] ; Mary,Canavan ^[1] ; Jean M,Fletcher ^[2] ; Ronan H,Mullan ^[1] ; Suzanne,Cole ^[2] ; Ling-Yang,Hao ^[1] ; Michael G,Monaghan ^[2] ; Sunil,Nagpal ^[1] ; Douglas J,Veale ^[2] ; Ursula,Fearon ^[3]

作者单位： Molecular Rheumatology, Clinical Medicine, Trinity Biomedical Science Institute, Dublin, Ireland. ^[1] Eular Centre for Arthritis and Rheumatic Diseases, St Vincent's University Hospital, Univeristy College Dublin, Dublin, Ireland. ^[2] Translational Immunology, Trinity Biomedical Sciences Institute, Trinity College Dublin, Dublin, Ireland. ^[3] Department of Mechanical and Manufacturing Engineering, Trinity College Dublin, Dublin, Ireland. ^[4] Immunology, Janssen Research & Development, Spring House, PA, USA. ^[5] Department of Rheumatology, Tallaght University Hospital, Trinity College Dublin, Dublin, Ireland. ^[6] Molecular Rheumatology, Clinical Medicine, Trinity Biomedical Science Institute, Dublin, Ireland Fearonu@tcd.ie. ^[7]

关键词 Arthritis, Psoriatic Arthritis, Rheumatoid Autoimmunity

DOI 10.1136/annrheumdis-2021-221761

PMID 35701153

发布时间 2024-05-14

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准确一般字义偏差语意偏离译文作用小提交