SARS-CoV-2 infection of human brain microvascular endothelial cells leads to inflammatory activation through NF-κB non-canonical pathway and mitochondrial remodeling.

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作者： Silvia,Torices ^[1] ; Carolline,Motta ^[2] ; Barbara,da Rosa ^[2] ; Anne,Marcos ^[2] ; Liandra,Alvarez-Rosa ^[2] ; Michele,Siqueira ^[3] ; Thaidy,Moreno-Rodriguez ^[4] ; Aline,Matos ^[5] ; Braulia,Caetano ^[2] ; Jessica,Martins ^[2] ; Luis,Gladulich ^[2] ; Erick,Loiola ^[6] ; Olivia,Bagshaw ^[7] ; Jeffrey,Stuart ^[7] ; Marilda,Siqueira ^[8] ; Joice,Stipursky ^[3] ; Michal,Toborek ^[9] ; Daniel,Adesse ^[10]

作者单位： University of Miami Miller School of Medicine. ^[1] Fundacao Oswaldo Cruz. ^[2] Universidade Federal do Rio de Janeiro. ^[3] University of California San Francisco. ^[4] Laboratório de Vírus Respiratório e do Sarampo, Instituto Oswaldo Cruz, Fundação Oswaldo Cruz, FIOCRUZ, Rio de Janeiro, Brazil. ^[5] Fluminense Federal University. ^[6] Brock University. ^[7] Oswaldo Cruz Foundation. ^[8] University of Miami. ^[9] Instituto Oswaldo Cruz. ^[10]

DOI 10.21203/rs.3.rs-1762855/v1

PMID 35734086

发布时间 2024-09-23

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SARS-CoV-2 infection of human brain microvascular endothelial cells leads to inflammatory activation through NF-κB non-canonical pathway and mitochondrial remodeling.

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SARS-CoV-2 infection of human brain microvascular endothelial cells leads to inflammatory activation through NF-κB non-canonical pathway and mitochondrial remodeling.

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