GenX induces fibroinflammatory gene expression in primary human hepatocytes.

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作者： Dakota R,Robarts ^[1] ; Kaitlyn K,Venneman ^[2] ; Sumedha,Gunewardena ^[3] ; Udayan,Apte ^[4]

作者单位： Department of Pharmacology, Toxicology and Therapeutics, University of Kansas Medical Center, Kansas City, KS, USA. Electronic address: drobarts@kumc.edu. ^[1] Department of Pharmacology, Toxicology and Therapeutics, University of Kansas Medical Center, Kansas City, KS, USA. Electronic address: kvenneman@kumc.edu. ^[2] Department of Molecular and Integrative Physiology, University of Kansas Medical Center, Kansas City, KS, USA. Electronic address: sgunewardena@kumc.edu. ^[3] Department of Pharmacology, Toxicology and Therapeutics, University of Kansas Medical Center, Kansas City, KS, USA. Electronic address: uapte@kumc.edu. ^[4]

关键词 GenX Inflammation PFAS Primary human hepatocytes Proliferation Toxicogenomics

主题词氟碳化合物(Fluorocarbons);基因表达(Gene Expression);肝细胞(Hepatocytes);人类(Humans);丙酸盐类(Propionates)

DOI 10.1016/j.tox.2022.153259

PMID 35850385

发布时间 2022-12-14

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