Loss of TIP60 (KAT5) abolishes H2AZ lysine 7 acetylation and causes p53, INK4A, and ARF-independent cell cycle arrest.

导出 OA

翻译打印收藏纠错

第一作者： Johannes,Wichmann

第一单位： Walter & Eliza Hall Institute of Medical Research, Melbourne, VIC, Australia.

作者： Johannes,Wichmann ^[1] ; Catherine,Pitt ^[2] ; Samantha,Eccles ^[1] ; Alexandra L,Garnham ^[2] ; Connie S N,Li-Wai-Suen ^[1] ; Rose,May ^[1] ; Elizabeth,Allan ^[2] ; Stephen,Wilcox ^[1] ; Marco J,Herold ^[2] ; Gordon K,Smyth ^[1] ; Brendon J,Monahan ^[1] ; Tim,Thomas ^[3] ; Anne K,Voss ^[1]

作者单位： Walter & Eliza Hall Institute of Medical Research, Melbourne, VIC, Australia. ^[1] Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia. ^[2] Cancer Therapeutics CRC, Parkville, VIC, Australia. ^[3] School of Mathematics and Statistics, University of Melbourne, Parkville, VIC, Australia. ^[4] Walter & Eliza Hall Institute of Medical Research, Melbourne, VIC, Australia. tthomas@wehi.edu.au. ^[5] Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia. tthomas@wehi.edu.au. ^[6] Walter & Eliza Hall Institute of Medical Research, Melbourne, VIC, Australia. avoss@wehi.edu.au. ^[7] Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia. avoss@wehi.edu.au. ^[8]