Angiotensin AT<sub>2</sub> Receptor Stimulation Alleviates Collagen-Induced Arthritis by Upregulation of Regulatory T Cell Numbers.

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第一作者： Bettina,Sehnert

第一单位： Department of Rheumatology and Clinical Immunology, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

作者： Bettina,Sehnert ^[1] ; Veronica,Valero-Esquitino ^[2] ; Georg,Schett ^[3] ; Thomas,Unger ^[4] ; Ulrike Muscha,Steckelings ^[5] ; Reinhard Edmund,Voll ^[6]

作者单位： Department of Rheumatology and Clinical Immunology, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany. ^[1] Center for Cardiovascular Research, Charité-Medical Faculty Berlin, Berlin, Germany. ^[2] Department of Internal Medicine 3 - Rheumatology and Immunology, Friedrich-Alexander-University Erlangen-Nürnberg (FAU) and Universitätsklinikum Erlangen, Erlangen, Germany.;Deutsches Zentrum Immuntherapie, Friedrich-Alexander-University Erlangen-Nürnberg (FAU) and Universitätsklinikum Erlangen, Erlangen, Germany. ^[3] Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, Netherlands. ^[4] Institute of Molecular Medicine (IMM) - Department of Cardiovascular & Renal Research, University of Southern Denmark, Odense, Denmark. ^[5] Department of Rheumatology and Clinical Immunology, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.;Center for Chronic Immunodeficiency (CCI) Freiburg, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany. ^[6]

关键词 angiotensin AT2 receptor agonist collagen-induced arthritis (CIA)cytokines regulatory T cells renin–angiotensin system