Role of Oxidative Stress in Cardiac Dysfunction and Subcellular Defects Due to Ischemia-Reperfusion Injury.

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作者： Naranjan S,Dhalla ^[1] ; Anureet K,Shah ^[2] ; Adriana,Adameova ^[3] ; Monika,Bartekova ^[4]

作者单位： St. Boniface Hospital Albrechtsen Research Centre, Department of Physiology and Pathophysiology, Institute of Cardiovascular Sciences, Max Rady College of Medicine, University of Manitoba, Winnipeg, MB R2H 2A6, Canada. ^[1] School of Kinesiology, Nutrition and Food Science, California State University, Los Angeles, CA 90032, USA. ^[2] Department of Pharmacology and Toxicology, Faculty of Pharmacy, Comenius University in Bratislava, Odbojarov 10, 832 32 Bratislava, Slovakia. ^[3] Centre of Experimental Medicine, Institute for Heart Research, Slovak Academy of Sciences, Dubravska Cesta 9, 841 04 Bratislava, Slovakia. ^[4]

关键词 Ca2+-handling abnormalities cardiac subcellular defects intracellular Ca2+-overload ischemia-reperfusion injury myocardial inflammation oxyradicals and antioxidants protease activation

DOI 10.3390/biomedicines10071473

PMID 35884777

发布时间 2022-07-31

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Biomedicines

2022年10卷7期

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Role of Oxidative Stress in Cardiac Dysfunction and Subcellular Defects Due to Ischemia-Reperfusion Injury.

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Role of Oxidative Stress in Cardiac Dysfunction and Subcellular Defects Due to Ischemia-Reperfusion Injury.

Biomedicines

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Biomedicines

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