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ER-Stress and Senescence Coordinately Promote Endothelial Barrier Dysfunction in Diabetes-Induced Atherosclerosis.

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第一作者： Sameen,Fatima

第一单位： Institute of Laboratory Medicine, Clinical Chemistry and Molecular Diagnostic, University Hospital, 04103 Leipzig, Germany.;Institute of Experimental Internal Medicine, Medical Faculty, Otto von Guericke University, 39120 Magdeburg, Germany.

作者： Sameen,Fatima ^[1] ; Saira,Ambreen ^[2] ; Akash,Mathew ^[2] ; Ahmed,Elwakiel ^[2] ; Anubhuti,Gupta ^[2] ; Kunal,Singh ^[2] ; Shruthi,Krishnan ^[2] ; Rajiv,Rana ^[2] ; Hamzah,Khawaja ^[2] ; Dheerendra,Gupta ^[2] ; Jayakumar,Manoharan ^[2] ; Christian,Besler ^[3] ; Ulrich,Laufs ^[4] ; Shrey,Kohli ^[2] ; Berend,Isermann ^[2] ; Khurrum,Shahzad ^[2]

作者单位： Institute of Laboratory Medicine, Clinical Chemistry and Molecular Diagnostic, University Hospital, 04103 Leipzig, Germany.;Institute of Experimental Internal Medicine, Medical Faculty, Otto von Guericke University, 39120 Magdeburg, Germany. ^[1] Institute of Laboratory Medicine, Clinical Chemistry and Molecular Diagnostic, University Hospital, 04103 Leipzig, Germany. ^[2] Cardiology, Leipzig Heart Center, University of Leipzig, 04289 Leipzig, Germany. ^[3] Klinik und Poliklinik für Kardiologie, University Hospital Leipzig, 04103 Leipzig, Germany. ^[4]

关键词 activated protein C atherosclerosis diabetes endothelial cells senescence unfolded protein response

主题词动物(Animals);动脉粥样硬化(Atherosclerosis);糖尿病(Diabetes Mellitus);葡萄糖(Glucose);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);蛋白质C(Protein C)

DOI 10.3390/nu14142786

PMID 35889743

发布时间 2025-10-31

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准确一般字义偏差语意偏离译文作用小提交