Synthetic Peucedanocoumarin IV Prevents α-Synuclein Neurotoxicity in an Animal Model of Parkinson's Disease.

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作者： Heejeong,Kim ^[1] ; Han-Joo,Maeng ^[2] ; Ji Hun,Kim ^[1] ; Jin-Ha,Yoon ^[2] ; Yohan,Oh ^[3] ; Seung-Mann,Paek ^[4] ; Yunjong,Lee ^[5]

作者单位： Department of Pharmacology, Samsung Biomedical Research Institute, Sungkyunkwan University School of Medicine, Suwon 16419, Korea. ^[1] College of Pharmacy, Gachon University, Incheon 21936, Korea. ^[2] Department of Biomedical Science, Graduate School of Biomedical Science and Engineering, Hanyang University, Seoul 04763, Korea. ^[3] Department of Biochemistry and Molecular Biology, College of Medicine, Hanyang University, Seoul 04763, Korea. ^[4] College of Pharmacy and Research Institute of Pharmaceutical Sciences, Gyeongsang National University, Jinju Daero 501, Jinju 52828, Korea. ^[5]

关键词 Parkinson’s disease dopaminergic cell loss organic synthesis peucedanocoumarin IV pharmacokinetics α-synuclein preformed fibril

主题词动物(Animals);香豆素类(Coumarins);疾病模型, 动物(Disease Models, Animal);小鼠(Mice);帕金森病(Parkinson Disease);大鼠(Rats);α突触核蛋白(alpha-Synuclein)

DOI 10.3390/ijms23158618

PMID 35955753

发布时间 2022-08-23

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