Fingolimod does not prevent syndecan-4 shedding from the endothelial glycocalyx in a cultured human umbilical vein endothelial cell model of vascular injury.

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作者： Elissa M,Milford ^[1] ; Lara,Meital ^[2] ; Anna,Kuballa ^[3] ; Michael C,Reade ^[4] ; Fraser D,Russell ^[3]

作者单位： Faculty of Medicine, University of Queensland, Herston, QLD, Australia. elissa.milford@defence.gov.au. ^[1] Intensive Care Unit, Royal Brisbane and Women's Hospital, Butterfield St., Herston, QLD, Australia. elissa.milford@defence.gov.au. ^[2] School of Health and Behavioural Sciences, University of the Sunshine Coast, Maroochydore, QLD, Australia. ^[3] Centre for Bioinnovation, University of the Sunshine Coast, Maroochydore, QLD, Australia. ^[4] Faculty of Medicine, University of Queensland, Herston, QLD, Australia. ^[5] Intensive Care Unit, Royal Brisbane and Women's Hospital, Butterfield St., Herston, QLD, Australia. ^[6] Joint Health Command, Australian Defence Force, Canberra, ACT, Australia. ^[7]

关键词 Cell culture Endothelium FTY720 Fingolimod Fresh frozen plasma Glycocalyx Human umbilical vein endothelial cells Sphingosine 1-phosphate Syndecan-1 Syndecan-4

DOI 10.1186/s40635-022-00462-7

PMID 35980492

发布时间 2022-08-29

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Intensive care medicine experimental

2022年10卷1期

34页

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Fingolimod does not prevent syndecan-4 shedding from the endothelial glycocalyx in a cultured human umbilical vein endothelial cell model of vascular injury.

Intensive care medicine experimental

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Intensive care medicine experimental

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Fingolimod does not prevent syndecan-4 shedding from the endothelial glycocalyx in a cultured human umbilical vein endothelial cell model of vascular injury.

Intensive care medicine experimental

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Intensive care medicine experimental

相关期刊

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