TOX deficiency facilitates the differentiation of IL-17A-producing γδ T cells to drive autoimmune hepatitis.

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作者： Qifeng,He ^[1] ; Yijun,Lu ^[1] ; Wenfang,Tian ^[1] ; Runqiu,Jiang ^[1] ; Weiwei,Yu ^[1] ; Yong,Liu ^[1] ; Meiling,Sun ^[1] ; Fei,Wang ^[1] ; Haitian,Zhang ^[1] ; Ning,Wu ^[2] ; Zhongjun,Dong ^[2] ; Beicheng,Sun ^[3]

作者单位： Department of Hepatobiliary Surgery, Affiliated Drum Tower Hospital, Medical School of Nanjing University, Nanjing, China. ^[1] Department of Hepatobiliary Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, China. ^[2] Department of Hepatobiliary Surgery, Affiliated Drum Tower Hospital, Medical School of Nanjing University, Nanjing, China. sunbc@nju.edu.cn. ^[3] Department of Hepatobiliary Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, China. sunbc@nju.edu.cn. ^[4]

关键词 Autoimmune hepatitis IL-17A Immune tolerance TOX γδ T cell

主题词动物(Animals);自身抗原(Autoantigens);肝炎, 自身免疫性(Hepatitis, Autoimmune);白细胞介素17(Interleukin-17);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);受体, 抗原, T细胞, γ-δ(Receptors, Antigen, T-Cell, gamma-delta);T淋巴细胞(T-Lymphocytes);胸腺(Thymus Gland)

DOI 10.1038/s41423-022-00912-y

PMID 35986136

发布时间 2023-10-02

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Cellular & molecular immunology

2022年19卷10期

1102-1116页

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TOX deficiency facilitates the differentiation of IL-17A-producing γδ T cells to drive autoimmune hepatitis.

Cellular & molecular immunology

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Cellular & molecular immunology

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TOX deficiency facilitates the differentiation of IL-17A-producing γδ T cells to drive autoimmune hepatitis.

Cellular & molecular immunology

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Cellular & molecular immunology

相关期刊

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