Faecal microbiota transplant ameliorates gut dysbiosis and cognitive deficits in Huntington's disease mice.

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作者： Carolina,Gubert ^[1] ; Jocelyn M,Choo ^[2] ; Chloe J,Love ^[1] ; Saritha,Kodikara ^[3] ; Bethany A,Masson ^[1] ; Jamie J M,Liew ^[1] ; Yiwen,Wang ^[3] ; Geraldine,Kong ^[1] ; Vinod K,Narayana ^[4] ; Thibault,Renoir ^[1] ; Kim Anh,Lê Cao ^[3] ; Geraint B,Rogers ^[2] ; Anthony J,Hannan ^[1]

作者单位： Florey Institute of Neuroscience and Mental Health, Melbourne Brain Centre, University of Melbourne, Parkville, VIC 3010, Australia. ^[1] Microbiome and Host Health, South Australian Health and Medical Research Institute, Adelaide, SA 5001, Australia. ^[2] Melbourne Integrative Genomics, School of Mathematics and Statistics, University of Melbourne, Parkville, VIC 3010, Australia. ^[3] Bio21 Institute and Department of Biochemistry and Molecular Biology, University of Melbourne, Parkville, VIC 3010, Australia. ^[4]

关键词 Huntington’s disease dementia gut microbiome microbiota–gut–brain axis neurodegenerative disorder

DOI 10.1093/braincomms/fcac205

PMID 36035436

发布时间 2022-08-30

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Brain communications

2022年4卷4期

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Faecal microbiota transplant ameliorates gut dysbiosis and cognitive deficits in Huntington's disease mice.

Brain communications

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Faecal microbiota transplant ameliorates gut dysbiosis and cognitive deficits in Huntington's disease mice.

Brain communications

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Brain communications

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