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首页> Atherosclerosis> Bves maintains vascular smooth muscle cell contractile phenotype and protects against transplant vasculopathy via Dusp1-dependent p38MAPK and ERK1/2 signaling.

Bves maintains vascular smooth muscle cell contractile phenotype and protects against transplant vasculopathy via Dusp1-dependent p38MAPK and ERK1/2 signaling.

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作者： Jin-Xin,Liu ^[1] ; Tong,Huang ^[2] ; Dawei,Xie ^[3] ; Qihong,Yu ^[4]

作者单位： Department of Otolaryngology-Head and Neck Surgery, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China. ^[1] The Eight Affiliated Hospital, Sun Yat-sen University, Shenzhen, China. ^[2] State Key Laboratory of Molecular Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China. ^[3] Hepatic Surgery Center, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China; Clinical Medical Research Center of Hepatic Surgery at Hubei Province, Wuhan, China. Electronic address: qhygdqk@hust.edu.cn. ^[4]

关键词 Bves Contractile phenotype Dusp1 Transplant vasculopathy VSMC

主题词动物(Animals);细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);双特异性磷酸酶1(Dual Specificity Phosphatase 1);MAP激酶信号系统(MAP Kinase Signaling System);肌, 平滑, 血管(Muscle, Smooth, Vascular);肌细胞, 平滑肌(Myocytes, Smooth Muscle);新内膜(Neointima);表型(Phenotype);大鼠(Rats);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)

DOI 10.1016/j.atherosclerosis.2022.08.010

PMID 36037759

发布时间 2022-10-13

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