The crosstalk between MYC and mTORC1 during osteoclastogenesis.

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作者： Seyeon,Bae ^[1] ; Brian,Oh ^[2] ; Jefferson,Tsai ^[1] ; Peter Sang Uk,Park ^[1] ; Matthew Blake,Greenblatt ^[1] ; Eugenia G,Giannopoulou ^[3] ; Kyung-Hyun,Park-Min ^[1]

作者单位： Arthritis and Tissue Degeneration Program, David Z. Rosensweig Genomics Research Center, Hospital for Special Surgery, New York, NY, United States. ^[1] Department of Medicine, Weill Cornell Medical College, New York, NY, United States. ^[2] Department of Pathology, Weill Cornell Medical College, New York, NY, United States. ^[3] Biological Sciences Department, New York City College of Technology, City University of New York, Brooklyn, NY, United States. ^[4] BCMB Allied Program, Weill Cornell Graduate School of Medical Sciences, New York, NY, United States. ^[5]

关键词 GADD34 (PPP1R15A)MYC (c-myc)bone resorption mTORC1 (mechanistic target of rapamycin complex 1)osteoclast (OC)

DOI 10.3389/fcell.2022.920683

PMID 36060812

发布时间 2024-05-25

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Frontiers in cell and developmental biology

2022年10卷

920683页

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The crosstalk between MYC and mTORC1 during osteoclastogenesis.

Frontiers in cell and developmental biology

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The crosstalk between MYC and mTORC1 during osteoclastogenesis.

Frontiers in cell and developmental biology

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Frontiers in cell and developmental biology

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