An ankylosing spondylitis risk variant alters osteoclast differentiation.

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作者： Fangyi,Wu ^[1] ; Xuling,Han ^[1] ; Jing,Liu ^[1] ; Zhenghua,Zhang ^[2] ; Kexiang,Yan ^[2] ; Beilan,Wang ^[3] ; Lin,Yang ^[3] ; Hejian,Zou ^[4] ; Chengde,Yang ^[5] ; Wei,Huang ^[3] ; Li,Jin ^[1] ; Jiucun,Wang ^[1] ; Feng,Qian ^[1] ; Zhenmin,Niu ^[6]

作者单位： State Key Laboratory of Genetic Engineering, Shanghai Public Health Clinical Center, Human Phenome Institute, Zhangjiang Fudan International Innovation Center and School of Life Sciences, Fudan University, Shanghai, China. ^[1] Division of Dermatology, Huashan Hospital, Fudan University, Shanghai, China. ^[2] Shanghai-MOST Key Laboratory of Health and Disease Genomics, Chinese National Human Genome Center at Shanghai and Shanghai Institute for Biomedical and Pharmaceutical Technologies, Shanghai, China. ^[3] Division of Rheumatology, Huashan Hospital, Fudan University, Shanghai, China. ^[4] Department of Rheumatology and Immunology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, China. ^[5] Ministry of Education Key Laboratory of Contemporary Anthropology, School of Life Sciences, Fudan University, Shanghai, China. ^[6]

关键词 AS PSMB1 RANKL signal osteoclast differentiation proteasome