首页> Journal of applied physiology (Bethesda, Md. : 1985)> LP07 and LLC preclinical models of lung cancer induce divergent anabolic deficits and expression of pro-inflammatory effectors of muscle wasting.

LP07 and LLC preclinical models of lung cancer induce divergent anabolic deficits and expression of pro-inflammatory effectors of muscle wasting.

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第一作者： Daniel J,Belcher

第一单位： Huck Institutes of the Life Sciences, The Pennsylvania State University, University Park, Pennsylvania.

作者： Daniel J,Belcher ^[1] ; Maria,Guitart ^[2] ; Brian,Hain ^[3] ; Hyo-Gun,Kim ^[4] ; David,Waning ^[5] ; Esther,Barreiro ^[2] ; Gustavo A,Nader ^[6]

作者单位： Huck Institutes of the Life Sciences, The Pennsylvania State University, University Park, Pennsylvania. ^[1] Pulmonology Department-Muscle Wasting and Cachexia in Chronic Respiratory Diseases and Lung Cancer Research Group, IMIM-Hospital del Mar, Barcelona, Spain.;Department of Medicine and Life Sciences (MELIS), Universitat Pompeu Fabra (UPF), Barcelona, Spain.;Network of Excellence in Lung Diseases (CIBERES), Instituto de Salud Carlos III (ISCIII), Barcelona, Spain. ^[2] Department of Cellular and Molecular Physiology, The Pennsylvania State University College of Medicine, Hershey, Pennsylvania. ^[3] Department of Kinesiology, The Pennsylvania State University, University Park, Pennsylvania. ^[4] Department of Cellular and Molecular Physiology, The Pennsylvania State University College of Medicine, Hershey, Pennsylvania.;Penn State Cancer Institute, The Pennsylvania State University, University Park, Pennsylvania. ^[5] Huck Institutes of the Life Sciences, The Pennsylvania State University, University Park, Pennsylvania.;Department of Kinesiology, The Pennsylvania State University, University Park, Pennsylvania.;Penn State Cancer Institute, The Pennsylvania State University, University Park, Pennsylvania. ^[6]

关键词 cachexia inflammation lung cancer muscle wasting ribosomal RNA