APOBEC3G protects the genome of human cultured cells and mice from radiation-induced damage.

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作者： Yelena,Britan-Rosich ^[1] ; Jing,Ma ^[2] ; Eran,Kotler ^[3] ; Faizan,Hassan ^[1] ; Alexander,Botvinnik ^[1] ; Yoav,Smith ^[4] ; Ofra,Moshel ^[5] ; Abed,Nasereddin ^[6] ; Gunjan,Sharma ^[1] ; Eli,Pikarsky ^[1] ; Susan,Ross ^[2] ; Moshe,Kotler ^[1]

作者单位： Department of Pathology and Immunology, The Lautenberg Center for Immunology and Cancer Research, The Hebrew University Hadassah Medical School, Jerusalem, Israel. ^[1] Department of Microbiology and Immunology, College of Medicine, University of Illinois Chicago, IL, USA. ^[2] Department of Genetics, Stanford University School of Medicine, CA, USA. ^[3] Genomic Data Analysis, Hadassah Medical School, Hebrew University, Jerusalem, Israel. ^[4] Core Research Facility, Institute for Drug Research, School of Pharmacy, Faculty of Medicine, The Hebrew University-Hadassah Medical School, Jerusalem, Israel. ^[5] Core Research Facility of the Hebrew University-Hadassah Medical School, Jerusalem, Israel. ^[6]

关键词 APOBEC3G DNA damage response DNA repair cytidine deaminase homologous recombination mouse non-homologous end joining

主题词人类(Humans);小鼠(Mice);动物(Animals);细胞系(Cell Line);诱变(Mutagenesis);免疫, 先天(Immunity, Innate);胞苷脱氨酶(Cytidine Deaminase)

DOI 10.1111/febs.16673

PMID 36325681

发布时间 2024-04-02

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