Lysosomal TMEM9-LAMTOR4-controlled mTOR signaling integrity is required for mammary tumorigenesis.

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作者： Shengzhe,Zhang ^[1] ; Sung Ho,Lee ^[1] ; Litong,Nie ^[1] ; Yuanjian,Huang ^[1] ; Gengyi,Zou ^[1] ; Youn-Sang,Jung ^[1] ; Sohee,Jun ^[1] ; Jie,Zhang ^[1] ; Esther M,Lien ^[1] ; Junjie,Chen ^[1] ; Jae-Il,Park ^[1]

作者单位： Department of Experimental Radiation Oncology, Division of Radiation Oncology, the University of Texas MD Anderson Cancer Center, Houston, TX, 77030, USA. ^[1] Graduate School of Biomedical Sciences, the University of Texas MD Anderson Cancer Center, Houston, TX, 77030, USA. ^[2] Program in Genetics and Epigenetics, the University of Texas MD Anderson Cancer Center, Houston, TX, 77030, USA. ^[3]

主题词人类(Humans);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);溶酶体(Lysosomes);膜蛋白质类(Membrane Proteins);鸟嘌呤核苷酸交换因子类(Guanine Nucleotide Exchange Factors)

DOI 10.1002/cac2.12382

PMID 36336962

发布时间 2023-02-02

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Cancer communications (London, England)

2023年43卷1期

159-163页

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Lysosomal TMEM9-LAMTOR4-controlled mTOR signaling integrity is required for mammary tumorigenesis.

Cancer communications (London, England)

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Cancer communications (London, England)

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Lysosomal TMEM9-LAMTOR4-controlled mTOR signaling integrity is required for mammary tumorigenesis.

Cancer communications (London, England)

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Cancer communications (London, England)

相关期刊

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