NAD<sup>+</sup> attenuates cardiac injury after myocardial infarction in diabetic mice through regulating alternative splicing of VEGF in macrophages.
第一作者:
Lei,Jiao
第一单位:
Department of Pharmacology (State-Province Key Laboratories of Biomedicine-Pharmaceutics of China, Key Laboratory of Cardiovascular Medicine Research, Ministry of Education), College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang 150081, PR China.
作者:
主题词
动物(Animals);小鼠(Mice);选择性剪接(Alternative Splicing);糖尿病(Diabetes Mellitus);内皮细胞(Endothelial Cells);巨噬细胞(Macrophages);心肌梗死(Myocardial Infarction);NAD(NAD);新生血管化, 病理性(Neovascularization, Pathologic);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1016/j.vph.2022.107126
PMID
36351515
发布时间
2023-01-23
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Vascular pharmacology
107126页
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