I<sub>KACh</sub> is constitutively active via PKC epsilon in aging mediated atrial fibrillation.

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第一作者： Mengmeng,Chang

第一单位： Department of Molecular Pharmacology and Physiology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA.

作者： Mengmeng,Chang ^[1] ; Kirin D,Gada ^[2] ; Bojjibabu,Chidipi ^[1] ; Athanasios,Tsalatsanis ^[3] ; Justin,Gibbons ^[4] ; Elizabeth,Remily-Wood ^[5] ; Diomedes E,Logothetis ^[2] ; Jenna,Oberstaller ^[4] ; Sami F,Noujaim ^[1]

作者单位： Department of Molecular Pharmacology and Physiology, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA. ^[1] Department of Pharmaceutical Sciences, School of Pharmacy and Pharmaceutical Science, Bouvé College of Health Sciences, Center for Drug Discovery, Northeastern University, Boston, MA 02115, USA. ^[2] College of Medicine Office of Research, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA. ^[3] Center for Global Health and Infectious Diseases Research and USF Genomics Program, College of Public Health, University of South Florida, Tampa, FL 33612, USA. ^[4] Department of Molecular Medicine, Morsani College of Medicine, University of South Florida, Tampa, FL 33612, USA. ^[5]

关键词 Animal physiology Biological sciences Proteomics Transcriptomics

DOI 10.1016/j.isci.2022.105442

PMID 36388956

发布时间 2024-06-03

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2022年25卷11期

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I<sub>KACh</sub> is constitutively active via PKC epsilon in aging mediated atrial fibrillation.

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I<sub>KACh</sub> is constitutively active via PKC epsilon in aging mediated atrial fibrillation.

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