Forced expression of the non-coding RNA miR-17∼92 restores activation and function in CD28-deficient CD4<sup>+</sup> T cells.

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作者： Marianne,Dölz ^[1] ; Marko,Hasiuk ^[2] ; John D,Gagnon ^[1] ; Mara,Kornete ^[2] ; Romina,Marone ^[3] ; Glenn,Bantug ^[4] ; Robin,Kageyama ^[1] ; Christoph,Hess ^[1] ; K Mark,Ansel ^[2] ; Denis,Seyres ^[1] ; Julien,Roux ^[3] ; Lukas T,Jeker ^[4]

作者单位： Department of Biomedicine, Basel University Hospital and University of Basel, Hebelstrasse 20, CH-4031 Basel, Switzerland. ^[1] Transplantation Immunology & Nephrology, Basel University Hospital, Petersgraben 4, CH-4031 Basel, Switzerland. ^[2] Department of Microbiology and Immunology, University of California, San Francisco, San Francisco, CA 94143, USA. ^[3] Sandler Asthma Basic Research Center, University of California, San Francisco, San Francisco, CA 94143, USA. ^[4] Department of Medicine - CITIID, University of Cambridge, Puddicombe Way, Cambridge CB2 0AW, UK. ^[5] Swiss Institute of Bioinformatics, Basel, Switzerland. ^[6]

关键词 Biological sciences immunology molecular mechanism of gene regulation

DOI 10.1016/j.isci.2022.105372

PMID 36388982

发布时间 2024-06-03

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Forced expression of the non-coding RNA miR-17∼92 restores activation and function in CD28-deficient CD4<sup>+</sup> T cells.

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Forced expression of the non-coding RNA miR-17∼92 restores activation and function in CD28-deficient CD4<sup>+</sup> T cells.

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