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Natural history and neurodevelopmental outcomes in perinatal stress induced hyperinsulinism.

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第一作者： Winnie M,Sigal

第一单位： Division of Endocrinology and Diabetes, Children's Hospital of Philadelphia, Philadelphia, PA, United States.;Department of Pediatrics, The Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, United States.

作者： Winnie M,Sigal ^[1] ; Ohoud,Alzahrani ^[2] ; Gabriela M,Guadalupe ^[2] ; Herodes,Guzman ^[2] ; Jerilynn,Radcliffe ^[3] ; Nina H,Thomas ^[4] ; Abbas F,Jawad ^[5] ; Diva D,De Leon ^[1]

作者单位： Division of Endocrinology and Diabetes, Children's Hospital of Philadelphia, Philadelphia, PA, United States.;Department of Pediatrics, The Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, United States. ^[1] Division of Endocrinology and Diabetes, Children's Hospital of Philadelphia, Philadelphia, PA, United States. ^[2] Behavioral Neuroscience Core, Center for Human Phenomic Science, The Children's Hospital of Philadelphia, Philadelphia, PA, United States.;Division of Developmental and Behavioral Pediatrics, The Children's Hospital of Philadelphia, Philadelphia, PA, United States. ^[3] Behavioral Neuroscience Core, Center for Human Phenomic Science, The Children's Hospital of Philadelphia, Philadelphia, PA, United States.;Division of Child and Adolescent Psychiatry, The Children's Hospital of Philadelphia, Philadelphia, PA, United States.;Department of Psychiatry, The Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, United States. ^[4] Department of Pediatrics, Children's Hospital of Philadelphia, Philadelphia, PA, United States.;Biostatistics Core, Center for Human Phenomic Science, The Children's Hospital of Philadelphia, Philadelphia, PA, United States. ^[5]

关键词 brain damage glucose hypoglycemia insulin neonates

DOI 10.3389/fped.2022.999274

PMID 36389353

发布时间 2022-11-19

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Natural history and neurodevelopmental outcomes in perinatal stress induced hyperinsulinism.

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Natural history and neurodevelopmental outcomes in perinatal stress induced hyperinsulinism.

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