Loss of pulsatility with continuous-flow left ventricular assist devices and the significance of the arterial endothelium in von-Willebrand factor production and degradation.

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作者： Guruprasad A,Giridharan ^[1] ; Ian C,Berg ^[2] ; Esraa,Ismail ^[3] ; Khanh T,Nguyen ^[2] ; Jana,Hecking ^[2] ; James K,Kirklin ^[4] ; Xuanhong,Cheng ^[5] ; Palaniappan,Sethu ^[2]

作者单位： Department of Bioengineering, J. B. Speed School of Engineering, University of Louisville, Louisville, Kentucky, USA. ^[1] Division of Cardiovascular Disease, Heersink School of Medicine, University of Alabama at Birmingham, Birmingham, Alabama, USA.;Department of Biomedical Engineering, School of Engineering and Heersink School of Medicine, University of Alabama at Birmingham, Birmingham, Alabama, USA. ^[2] Department of Bioengineering, Lehigh University, Bethlehem, Pennsylvania, USA. ^[3] Division of Cardiothoracic Surgery, Department of Surgery, Heersink School of Medicine, University of Alabama at Birmingham, Birmingham, Alabama, USA. ^[4] Department of Bioengineering, Lehigh University, Bethlehem, Pennsylvania, USA.;Department of Materials Science and Engineering, Lehigh University, Bethlehem, Pennsylvania, USA. ^[5]

关键词 acquired von Willebrand syndrome continuous flow loss of pulsatility von Willebrand Factor

主题词人类(Humans);von Willebrand因子(von Willebrand Factor);心脏辅助装置(Heart-Assist Devices);内皮细胞(Endothelial Cells);出血(Hemorrhage);血管性血友病(von Willebrand Diseases);内皮(Endothelium)

DOI 10.1111/aor.14456

PMID 36404709

发布时间 2024-09-23

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