Nonreceptor Tyrosine Kinase c-Abl-Mediated PHB2 Phosphorylation Aggravates Mitophagy Disorder in Parkinson's Disease Model.

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第一作者： Yongjiang,Zhang

第一单位： Neuromolecular Biology Laboratory, The First Affiliated Hospital, College of Clinical Medicine of Henan University of Science and Technology, Luoyang 471003, China.

作者： Yongjiang,Zhang ^[1] ; Jiannan,Wu ^[1] ; Weina,Jin ^[2] ; Mengmeng,Shen ^[1] ; Shiyi,Yin ^[1] ; Xiaoyi,Lai ^[1] ; Hongxia,Ma ^[1] ; Menghan,Jiang ^[3] ; Dongxue,Sun ^[1] ; Junqiang,Yan ^[3]

作者单位： Neuromolecular Biology Laboratory, The First Affiliated Hospital, College of Clinical Medicine of Henan University of Science and Technology, Luoyang 471003, China. ^[1] China National Clinical Research Center for Neurological Diseases, Jing-Jin Center for Neuroinflammation, Beijing Tiantan Hospital, Capital Medical University, Beijing 100000, China. ^[2] Department of Neurology, The First Affiliated Hospital, College of Clinical Medicine of Henan University of Science and Technology, Luoyang 471003, China. ^[3]

主题词动物(Animals);人类(Humans);小鼠(Mice);磷酰化(Phosphorylation);帕金森病(Parkinson Disease);TYK2激酶(TYK2 Kinase);神经母细胞瘤(Neuroblastoma);1-甲基-4-苯基吡啶(1-Methyl-4-phenylpyridinium);酪氨酸(Tyrosine)

DOI 10.1155/2022/9233749

PMID 36406767

发布时间 2022-11-22

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Oxidative medicine and cellular longevity

2022年2022卷

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Nonreceptor Tyrosine Kinase c-Abl-Mediated PHB2 Phosphorylation Aggravates Mitophagy Disorder in Parkinson's Disease Model.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

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Nonreceptor Tyrosine Kinase c-Abl-Mediated PHB2 Phosphorylation Aggravates Mitophagy Disorder in Parkinson's Disease Model.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

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