CircCAMTA1 facilitates atrial fibrosis by regulating the miR-214-3p/TGFBR1 axis in atrial fibrillation.

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第一作者： Li,Zhang

第一单位： Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, Youzheng Street No. 23, Nangang District, 150001, Harbin, Heilongjiang, China.

作者： Li,Zhang ^[1] ; Qi,Lou ^[1] ; Wei,Zhang ^[2] ; Wen,Yang ^[1] ; Luyifei,Li ^[1] ; Hongyan,Zhao ^[3] ; Yihui,Kong ^[1] ; Weimin,Li ^[4]

作者单位： Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, Youzheng Street No. 23, Nangang District, 150001, Harbin, Heilongjiang, China. ^[1] Department of Cardiovascular Surgery, The First Affiliated Hospital of Harbin Medical University, Youzheng Street No. 23, Nangang District, 150001, Harbin, Heilongjiang, China. ^[2] Department of Cardiology, The People's Hospital of Liaoning Province, Wenyi Road No. 33, Shenhe District, 110000, Shenyang, Liaoning, China. ^[3] Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, Youzheng Street No. 23, Nangang District, 150001, Harbin, Heilongjiang, China. liweimin_2009@163.com. ^[4]

关键词 Atrial fibrillation Atrial fibroblast Transforming growth factor β receptor 1 angiotensin-II circCAMTA1 miR-214-3p

主题词人类(Humans);心房颤动(Atrial Fibrillation);微RNAs(MicroRNAs);纤维化(Fibrosis);胶原(Collagen);成纤维细胞(Fibroblasts);血管紧张素Ⅱ(Angiotensin II)

DOI 10.1007/s10735-022-10110-9

PMID 36417034

发布时间 2023-02-10

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