Dual Targeting of MDM4 and FTH1 by MMRi71 for Induced Protein Degradation and p53-Independent Apoptosis in Leukemia Cells.

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作者： Rati,Lama ^[1] ; Samuel L,Galster ^[2] ; Chao,Xu ^[1] ; Luke W,Davison ^[2] ; Sherry R,Chemler ^[2] ; Xinjiang,Wang ^[1]

作者单位： Department of Pharmacology and Therapeutics, Roswell Park Comprehensive Cancer Center, Buffalo, NY 14263, USA. ^[1] Department of Chemistry, University at Buffalo, State University of New York, Buffalo, NY 14260, USA. ^[2]

关键词 E3 ligase FTH1 MDM4 MMRi apoptosis leukemia p53 quinolinol ubiquitination

主题词人类(Humans);原癌基因蛋白质c-mdm2(Proto-Oncogene Proteins c-mdm2);原癌基因蛋白质类(Proto-Oncogene Proteins);泛素蛋白连接酶类(Ubiquitin-Protein Ligases);细胞凋亡(Apoptosis);白血病(Leukemia);肿瘤(Neoplasms);细胞周期蛋白质类(Cell Cycle Proteins);铁蛋白质类(Ferritins);氧化还原酶类(Oxidoreductases)

DOI 10.3390/molecules27227665

PMID 36431769

发布时间 2023-06-13

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Molecules (Basel, Switzerland)

2022年27卷22期

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Dual Targeting of MDM4 and FTH1 by MMRi71 for Induced Protein Degradation and p53-Independent Apoptosis in Leukemia Cells.

Molecules (Basel, Switzerland)

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Molecules (Basel, Switzerland)

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Dual Targeting of MDM4 and FTH1 by MMRi71 for Induced Protein Degradation and p53-Independent Apoptosis in Leukemia Cells.

Molecules (Basel, Switzerland)

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Molecules (Basel, Switzerland)

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