Longitudinal analysis of invariant natural killer T cell activation reveals a cMAF-associated transcriptional state of NKT10 cells.

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第一作者： Harry,Kane

第一单位： Trinity Biomedical Science Institute, Trinity College Dublin, Dublin, Ireland.

作者： Harry,Kane ^[1] ; Nelson M,LaMarche ^[2] ; Áine,Ní Scannail ^[3] ; Amanda E,Garza ^[3] ; Hui-Fern,Koay ^[4] ; Adiba I,Azad ^[3] ; Britta,Kunkemoeller ^[3] ; Brenneth,Stevens ^[5] ; Michael B,Brenner ^[2] ; Lydia,Lynch ^[5]

作者单位： Trinity Biomedical Science Institute, Trinity College Dublin, Dublin, Ireland. ^[1] Division of Rheumatology, Inflammation, and Immunity, Brigham and Women's Hospital, Harvard Medical School, Boston, United States. ^[2] Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women's Hospital, Harvard Medical School, Boston, United States. ^[3] Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women's Hospital, Harvard Medical School, Boston, United States.;Department of Microbiology and Immunology, Peter Doherty Institute for Infection and Immunity, University of Melbourne, Melbourne, Australia. ^[4] Trinity Biomedical Science Institute, Trinity College Dublin, Dublin, Ireland.;Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women's Hospital, Harvard Medical School, Boston, United States. ^[5]

关键词 activation adipose tissue cellular metabolism computational biology human iNKT cells immunology inflammation mouse scRNA-Seq systems biology transcriptional remodeling

主题词动物(Animals);人类(Humans);小鼠(Mice);基因表达调控(Gene Expression Regulation);淋巴细胞活化(Lymphocyte Activation);自然杀伤T细胞(Natural Killer T-Cells)

DOI 10.7554/eLife.76586

PMID 36458691

发布时间 2024-12-02

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