Laminarin ameliorates alcohol-induced liver damage and its molecular mechanism in mice.

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第一作者： Tianyi,Guo

第一单位： Hunan Key Laboratory of Grain-oil Deep Process and Quality Control, Hunan Key Laboratory of Forestry Edible Resources Safety and Processing, Central South University of Forestry and Technology, Changsha, China.

作者： Tianyi,Guo ^[1] ; Lingfeng,Zhu ^[1] ; Yaping,Zhou ^[2] ; Shuai,Han ^[1] ; Yunyun,Cao ^[1] ; Zuomin,Hu ^[1] ; Yi,Luo ^[1] ; Liyuan,Bao ^[3] ; Xiuxiu,Wu ^[4] ; Dandan,Qin ^[1] ; Qinlu,Lin ^[1] ; Feijun,Luo ^[1]

作者单位： Hunan Key Laboratory of Grain-oil Deep Process and Quality Control, Hunan Key Laboratory of Forestry Edible Resources Safety and Processing, Central South University of Forestry and Technology, Changsha, China. ^[1] Hunan Agricultural Product Processing Institute, Hunan Academy of Agricultural Sciences, Changsha, China. ^[2] Department of Clinic Medicine, Xiangya School of Medicine, Central South University, Changsha, China. ^[3] Department of logistics, Changsha University, Changsha, China. ^[4]

关键词 alcoholic liver disease laminarin molecular mechanisms pathways

主题词小鼠(Mice);动物(Animals);维生素A(Vitamin A);肝疾病, 酒精性(Liver Diseases, Alcoholic);乙醇(Ethanol);谷胱甘肽(Glutathione);β葡聚糖类(beta-Glucans)

DOI 10.1111/jfbc.14500

PMID 36515171

发布时间 2023-01-13

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