Type I interferon signaling in SARS-CoV-2 associated neurocognitive disorder (SAND): Mapping host-virus interactions to an etiopathogenesis.

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作者： George D,Vavougios ^[1] ; Gabriel A,de Erausquin ^[2] ; Heather M,Snyder ^[3]

作者单位： Department of Neurology, University of Cyprus, Lefkosia, Cyprus. ^[1] Department of Respiratory Medicine, University of Thessaly, Larisa, Greece. ^[2] The Glenn Biggs Institute for Alzheimer's and Neurodegenerative Diseases, UTHSA, San Antonio, TX, United States. ^[3] Division of Medical and Scientific Relations, Alzheimer's Association, Chicago, IL, United States. ^[4]

关键词 SARS-CoV-2 cognitive impairment dementia host-virus interaction tauopathy type I interferon signaling

DOI 10.3389/fneur.2022.1063298

PMID 36570454

发布时间 2023-06-16

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Frontiers in neurology

2022年13卷

1063298页

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Type I interferon signaling in SARS-CoV-2 associated neurocognitive disorder (SAND): Mapping host-virus interactions to an etiopathogenesis.

Frontiers in neurology

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Frontiers in neurology

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Type I interferon signaling in SARS-CoV-2 associated neurocognitive disorder (SAND): Mapping host-virus interactions to an etiopathogenesis.

Frontiers in neurology

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Frontiers in neurology

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