Hypercapnia alters stroma-derived Wnt production to limit β-catenin signaling and proliferation in AT2 cells.

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作者： Laura A,Dada ^[1] ; Lynn C,Welch ^[1] ; Natalia D,Magnani ^[1] ; Ziyou,Ren ^[1] ; Hyebin,Han ^[1] ; Patricia L,Brazee ^[1] ; Diego,Celli ^[1] ; Annette S,Flozak ^[1] ; Anthea,Weng ^[1] ; Mariana Maciel,Herrerias ^[1] ; Vitalii,Kryvenko ^[2] ; István,Vadász ^[3] ; Constance E,Runyan ^[2] ; Hiam,Abdala-Valencia ^[3] ; Masahiko,Shigemura ^[1] ; S Marina,Casalino-Matsuda ^[1] ; Alexander V,Misharin ^[1] ; G R Scott,Budinger ^[1] ; Cara J,Gottardi ^[1] ; Jacob I,Sznajder ^[1]

作者单位： Pulmonary and Critical Care Medicine, Northwestern Feinberg School of Medicine, Chicago, Illinois, USA. ^[1] Justus Liebig University, Universities of Giessen and Marburg Lung Center, Member of the German Center for Lung Research, Department of Internal Medicine, Giessen, Germany. ^[2] The Cardio-Pulmonary Institute, Giessen, Germany. ^[3]

关键词 Cell Biology Pulmonology Respiration

主题词小鼠(Mice);β连环素(beta Catenin);细胞增殖(Cell Proliferation);高碳酸血(Hypercapnia);动物(Animals)

DOI 10.1172/jci.insight.159331

PMID 36626234

发布时间 2024-04-28

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2023年8卷4期

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Hypercapnia alters stroma-derived Wnt production to limit β-catenin signaling and proliferation in AT2 cells.

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Hypercapnia alters stroma-derived Wnt production to limit β-catenin signaling and proliferation in AT2 cells.

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