Microglia rely on SYK signalling to mount neuroprotective responses in models of Alzheimer's disease and multiple sclerosis.

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作者： Hannah,Ennerfelt ^[1] ; John R,Lukens ^[2]

作者单位： Department of Neurology, Stanford University, Palo Alto, CA, USA. ^[1] Center for Brain Immunology and Glia (BIG), Department of Neuroscience, University of Virginia, Charlottesville, VA, USA. ^[2]

关键词 Alzheimer's disease amyloid beta microglia multiple sclerosis myelin debris neurodegenerative disease neuroimmunology

主题词人类(Humans);阿尔茨海默病(Alzheimer Disease);多发性硬化(Multiple Sclerosis);小神经胶质细胞(Microglia);信号传导(Signal Transduction);脑(Brain)

DOI 10.1002/ctm2.1178

PMID 36629045

发布时间 2023-11-06

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Clinical and translational medicine

2023年13卷1期

e1178页

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Microglia rely on SYK signalling to mount neuroprotective responses in models of Alzheimer's disease and multiple sclerosis.

Clinical and translational medicine

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Clinical and translational medicine

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Microglia rely on SYK signalling to mount neuroprotective responses in models of Alzheimer's disease and multiple sclerosis.

Clinical and translational medicine

相似文献

Clinical and translational medicine

相关期刊

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