The PSEN1 E280G mutation leads to increased amyloid-β43 production in induced pluripotent stem cell neurons and deposition in brain tissue.

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作者： Nanet,Willumsen ^[1] ; Charles,Arber ^[2] ; Christopher,Lovejoy ^[1] ; Jamie,Toombs ^[1] ; Argyro,Alatza ^[1] ; Philip S J,Weston ^[3] ; Lucia,Chávez-Gutiérrez ^[1] ; John,Hardy ^[1] ; Henrik,Zetterberg ^[4] ; Nick C,Fox ^[5] ; Natalie S,Ryan ^[6] ; Tammaryn,Lashley ^[1] ; Selina,Wray ^[3]

作者单位： Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, London WC1N 1PJ, UK. ^[1] The Queen Square Brain Bank for Neurological Disorders, Department of Clinical and Movement Neuroscience, UCL Queen Square Institute of Neurology, London WC1N 1PJ, UK. ^[2] UK Dementia Research Institute, University College London, London WC1E 6AU, UK. ^[3] Dementia Research Centre, UCL Queen Square Institute of Neurology, London WC1E 6BT, UK. ^[4] VIB Center for Brain and Disease Research, 3000 Leuven, Belgium. ^[5] Department of Neurology, KU Leuven, 3000 Leuven, Belgium. ^[6] Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, Sahlgrenska Academy at University of Gothenburg, S-431 80 Mölndal, Sweden. ^[7]

关键词 APP Alzheimer’s disease amyloid-β induced pluripotent stem cells γ-secretase

DOI 10.1093/braincomms/fcac321

PMID 36687397

发布时间 2023-08-16

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Brain communications

2023年5卷1期

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The PSEN1 E280G mutation leads to increased amyloid-β43 production in induced pluripotent stem cell neurons and deposition in brain tissue.

Brain communications

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Brain communications

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The PSEN1 E280G mutation leads to increased amyloid-β43 production in induced pluripotent stem cell neurons and deposition in brain tissue.

Brain communications

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Brain communications

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