BMAL1 collaborates with CLOCK to directly promote DNA double-strand break repair and tumor chemoresistance.

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作者： Canfeng,Zhang ^[1] ; Liping,Chen ^[2] ; Lu,Sun ^[3] ; Heping,Jin ^[4] ; Kai,Ren ^[5] ; Shiqi,Liu ^[3] ; Yongyu,Qian ^[4] ; Shupeng,Li ^[5] ; Fangping,Li ^[4] ; Chengming,Zhu ^[5] ; Yong,Zhao ^[6] ; Haiying,Liu ^[3] ; Yan,Liu ^[3]

作者单位： The Seventh Affiliated Hospital, Sun Yat-Sen University, Shenzhen, 518107, China. zhangcf6@mail2.sysu.edu.cn. ^[1] The Center for Medical Research, The First People's Hospital of Nanning City, Nanning, 530021, China. ^[2] The Seventh Affiliated Hospital, Sun Yat-Sen University, Shenzhen, 518107, China. ^[3] MOE Key Laboratory of Gene Function and Regulation, School of Life Sciences, Sun Yat-sen University, Guangzhou, 510006, China. ^[4] State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, 510006, China. ^[5] State Key Laboratory of Chemical Oncogenomics, School of Chemical Biology and Biotechnology, Peking University Shenzhen Graduate School, Shenzhen, 518055, China. ^[6] The Seventh Affiliated Hospital, Sun Yat-Sen University, Shenzhen, 518107, China. liuyan92@mail.sysu.edu.cn. ^[7]