25-Hydroxycholesterol exacerbates vascular leak during acute lung injury.

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作者： Jennifer H,Madenspacher ^[1] ; Eric D,Morrell ^[2] ; Jeffrey G,McDonald ^[3] ; Bonne M,Thompson ^[4] ; Yue,Li ^[3] ; Konstantin G,Birukov ^[5] ; Anna A,Birukova ^[5] ; Renee D,Stapleton ^[5] ; Aidin,Alejo ^[6] ; Peer W,Karmaus ^[1] ; Julie M,Meacham ^[1] ; Prashant,Rai ^[1] ; Carmen,Mikacenic ^[1] ; Mark M,Wurfel ^[2] ; Michael B,Fessler ^[2]

作者单位： Immunity, Inflammation, and Disease Laboratory, National Institute of Environmental Health Sciences, NIH, Research Triangle Park, North Carolina, USA. ^[1] Section of Pulmonary, Critical Care, and Sleep Medicine, Harborview Medical Center, Seattle, Washington, USA. ^[2] Center for Human Nutrition and. ^[3] Department of Molecular Genetics, University of Texas Southwestern Medical Center, Dallas, Texas, USA. ^[4] Department of Anesthesiology, University of Maryland School of Medicine, Baltimore, Maryland, USA. ^[5] Department of Medicine, Larner College of Medicine, University of Vermont, Burlington, Vermont, USA. ^[6]

关键词 Cell stress Inflammation Innate immunity Mouse models Pulmonology

主题词动物(Animals);小鼠(Mice);羟基胆甾醇类(Hydroxycholesterols);巨噬细胞, 肺泡(Macrophages, Alveolar);急性肺损伤(Acute Lung Injury)

DOI 10.1172/jci.insight.155448

PMID 36821369

发布时间 2024-05-26

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25-Hydroxycholesterol exacerbates vascular leak during acute lung injury.

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